By Y. Reto. Clarion University.
Andersson GB (1981) Epidemiologic aspects on low-back pain in of sport selected is not ultimately important 160 mg super p-force free shipping, activities in industry 160 mg super p-force sale. Hu- best, of course, although other ball-based sports such as ber, Bern 72 3. Seated postures and sitting aids: a upright seated posture; b drooping seated posture; c kyphotic seated posture; d influence of writing height and slope of the writing surface on seated posture; e ball chair 3 a b c d e 3. Ihme N, Olszynska B, Lorani A, Weiss C, Kochs A (2002) Zusam- » While her elegance in ballet may appeal, menhang der vermehrten Innenrotation im Hüftgelenk mit einer the risk of scoliosis is very real. Epidemiologic Condition involving lateral bending of the spine of >10° aspects and work-related factors in the steel industry. McMaster MJ (1983) Infantile idiopathic scoliosis: Can it be pre- A rare form in which the deformity starts as early vented? J Bone Joint Surg (Am) 65: 612–7 as infancy or childhood (infantile or juvenile sco- 7. Rohrer MH, Santos-Eggimann B, Paccaud F, Haller-Maslov E (1994) Epidemiologic study of low back pain in 1398 Swiss con- liosis). Boys and girls are equally affected by this scripts between and 1985 and 1992. Saunders, Philadel- have their convexity to the left and are associated phia with kyphosis. Toroptsova NV, Benevolenskaya LI, Karyakin AN, Sergeev IL, ▬ The more common adolescent form starts during pu- Erdesz S (1995) »Cross-sectional« study of low back pain among workers at an industrial enterprise in Russia. The characteristic Classification by age at onset (according to the Ameri- features of infantile scoliosis differ from those of the ado- can Scoliosis Research Society): lescent form to such an extent that it can clearly be con- ▬ Infantile: 0–3 years sidered as a different disease. The prognosis for infantile ▬ Juvenile: 4–10 years scoliosis is very poor. Despite brace treatment, it will often ▬ Adolescent over 10 years old undergo substantial progression, resulting in the need for surgery even at an early age in many cases. Because juvenile scolioses are extremely rare (and do not behave according to a typical pattern), the British Juvenile scoliosis Scoliosis Society classifies only two entities: If the scoliosis occurs between the ages of 4 and 10, the ▬ Early onset: 0–7 years juvenile form is considered to be present. Girls are only ▬ Late onset over 7 years old slightly more frequently affected than boys. In addition to the thoracic location, lumbar and S-shaped curves also occur. Only 5% of scolioses are The condition known as resolving infantile scoliosis is not non-progressive, while the rest increase annually by 1–3° classed as an idiopathic scoliosis but is a special type of up to aged 10, and by 5–10° a year during the pubertal scoliotic posture. Adolescent (late onset) idiopathic scoliosis Resolving infantile scoliosis Resolving infantile scoliosis occurs at the age of a few This is by far the commonest form of scoliosis and is months, but has become relatively rare in the west as a characterized by the following features: result of the frequent use of the prone position. Resolving It is usually located at the thoracic level and almost infantile scoliosis is characterized by a long, usually left- without exception involves a right-convex curve. It occurs less commonly at the thoracolumbar The rib vertebral angle difference (RVAD) according to and lumbar levels, and such cases show a marked Mehta is measured to distinguish it from progressive tendency to go out of alignment. The prognosis is good scolioses are not truly idiopathic but occur second- and a spontaneous recovery can be expected in over 96% arily to leg length discrepancies or a lumbosacral of cases. In 76% of cases the scoliosis is left convex and often scolioses of the same severity. In the infant, an rib vertebral It is almost always associated with relative lordosis angle difference according to Mehta of more than 20° (for the thoracic level, an overall kyphotic angle of indicates that the condition is not the benign resolving less than 20° is considered to be relative lordosis). In scolioses in the infant, the diminished growth of the posterior sections forces angle between a vertical line passing through the vertebral body and the vertebral bodies to deviate laterally and to ro- the axis of the rib is measured on both the convex and concave sides. Instead of a scoliosis, one might describe this If the difference(γ)between the two angles is 20° or more, the scoliosis is very likely to be the progressive form rather than a spontaneously as a rotational lordosis. In adolescent scoliosis there is a disparity be- tween the growth of the vertebral bodies anteriorly and that of the posterior elements. The vertebral bodies grow faster than the posterior elements, resulting primarily in a lordosis. The diminished dorsal growth impedes the 3 ventrally located vertebral bodies from increasing in height, forcing them to become distorted, i. The idea of looking at in this way dates back to Somerville, and many more recent studies have con- firmed this theory [32, 59, 78, 93]. Lordosis is almost al- ways present in adolescent scoliosis, even when the spine appears kyphotic on the x-ray in a particular projection. It is conceivable that the spinal cord is protecting itself against the stretching stimulus of growth. Several investigations in recent years have reported the existence of intraspinal anomalies or neurological problems in a certain propor- tion of »idiopathic« scolioses. MRI studies have shown an intraspinal syrinx in 8% of typical idiopathic thoracic adolescent scolioses [23, 91]. Other investigators have found pathological somatosensory potentials in over 50 percent of cases of idiopathic adolescent scoliosis. Anteroposterior and lateral x-ray of an idiopathic thoracic does not appear to be relevant to such findings. Note the pronounced thoracic »handedness« responsible for the direction of the lateral lordosis a b c d ⊡ Fig. Principle of the development of idiopathic adolescent from a rod (a, c).
Such infections often Long-term observations have shown that patients can occur only at a late stage (i discount 160 mg super p-force with mastercard. An remain symptom-free for decades after correctly-per- increased risk exists for patients with severe acne generic super p-force 160 mg. It is We treat any patients diagnosed with acne with antibi- important that the spine should remain in alignment. Prolonged drainage is sometimes re- Fusion should not continue beyond the end vertebra quired, although an uncomplicated recovery invari- in the ventral derotation spondylodesis, in contrast ably occurs in young healthy patients. We therefore always ▬ A chylothorax can occur after anterior procedures instrument and fuse idiopathic lumbar scolioses from. In our own experience, spontaneous reabsorp- the anterior side and no lower than L3. Particularly Late complications serious problems can be expected in connection with ▬ A particular problem is posed by postoperative de- decompensation of the spine. This is observed more there is an especially great need for a treatment meth- frequently in cases involving the use of the Cotrel- od that avoids stiffening of the affected section of the Dubousset instrumentation for King type II scolioses spine. In our hospital we are currently developing a (corresponding to Lenke 2C or 3C) [57, 89, 110]. This method that allows correction of the scoliosis via an complication is thought to be attributable to exces- externally extendable implant. Whether this method sive rotation of the lumbar countercurve during the will one day be able to help avoid stiffening of the sco- maneuver for derotation of the thoracic spine. This liotic section of the spine in children and adolescents problem occurs much less frequently with the USS remains to be seen. Our therapeutic strategy for idiopathic adolescent ▬ Another serious problem is the loss of lumbar lordosis. Treatment concept for idiopathic scoliosis Growth phase Scoliosis angle less than 20° No treatment Scoliosis angle 20°–30° If progression is conformed and more than a year of the pubertal growth spurt remains (in girls up to a year after the menarche or Risser III) pre- 3 scribe brace, SpineCor treatment or Schroth therapy Scoliosis angle 30°–40° (thoracic) and 30°–50° lumbar If more than a year of the pubertal growth spurt remains: brace treat- ment Scoliosis angle from 40° (thoracic) and from 50° lumbar Surgery usually indicated. In case of decompensation (spine out of align ment) in thoracolumbar or lumbar scolioses sometimes also with smaller angles After completion of growth Scoliosis angle less than 40° (thoracic) or 60° lumbar No treatment Scoliosis angle 40°–60° Thoracic: surgery if the patient wants cosmetic improvement Lumbar: surgery recommended only if decompensation is present Scoliosis angle greater than 60° Surgery recommended since further progression is likely during adult- hood References 1. Connolly PJ, Von Schroeder HP, Johnson GE, Kostuik JP (1995) Ado- (2005) Dual growing rod technique for the treatment of progres- lescent idiopathic scoliosis. Long-term effect of instrumentation sive early-onset scoliosis: a multicenter study. Cotrel Y, Dubousset J (1984) Nouvelle technique d’osteosynthese weaning might reduce the psychological strain of Boston bracing: rachidienne segmentaire par voie posterieure. Rev Chir Orthop 70: a study of 136 patients with adolescent idiopathic scoliosis at 3. Danielsson AJ, Nachemson AL (2001) Radiologic findings and 96–9 curve progression 22 years after treatment for adolescent idio- 3. Ascani E, Bartolozzi P, Logroscino CA, Marchetti PG, Ponte A, Savini pathic scoliosis: comparison of brace and surgical treatment with R (1986) Natural history of untreated idiopathic scoliosis after skel- matching control group of straight individuals. Berlet G, Boubez G, Gurr K, Bailey S (1999) The USS pedicle hook gression, and sexual function in women 22 years after treatment system: a morphometric analysis of its safety in the thoracic spine. De Hart MM, Lauerman WC, Conely AH, Roettger RH, West JL, Cain body stapling procedure for the treatment of scoliosis in the JE (1994) Management of retroperitoneal chylous leakage. Bridwell K, Lenke L, Baldus C, Blanke K (1998) Major intraoperative J, Forriol F, Cara JA (eds) Basic research and clinical concepts. Pam- neurologic deficits in pediatric and adult spinal deformity patients. University of Navarra Medical School Incidence and etiology at one institution. Carr AJ, Jefferson RJ, Turner-Smith AR (1993) Family stature in patients with adolescent idiopathic scoliosis. Do T, Fras C, Burke S, Widmann R, Rawlins B, Boachie-Adjei O lescent idiopathic scoliosis: a histomorphometric study. Spine 26: (2001) Clinical value of routine preoperative magnetic resonance p19–23 imaging in adolescent idiopathic scoliosis. Clark C, Shufflebarger H (1999) Late-developing infection in in- three hundred and twenty-seven patients. Drerup B, Ellger B, Meyer zu Bentrup F, Hierholzer E (2001) Ras- course lectures, the American Academy of Orthopaedic Surgeons, terstereographische Funktionsaufnahmen. Edwards, Ann Arbor Mich zur biomechanischen Analyse der Skelettgeometrie. Coillard C, Leroux M, Zabjek K, Rivard C (2003) SpineCor–a non- 30:242–50 rigid brace for the treatment of idiopathic scoliosis: post-treat- 25. Dubousset J, Herring JA, Shufflebarger H (1989) The crankshaft ment results. Hopf A, Eysel P, Dubousset J (1995) CDH: preliminary report on a to scoliosis-preliminary report. Hung VW, Qin L, Cheung CS, Lam TP, Ng BK, Tse YK, Guo X, Lee KM, bility of anterior thoracoscopic spine surgery in children under 30 Cheng JC (2005) Osteopenia: a new prognostic factor of curve kilograms. Ebara S, Kamimura M, Itoh H, Kinoshita T, Takahashi J, Takaoka K, Am 87: 2709-16 Ohtsuka K (2000) A new system for the anterior restoration and 47. Jackson RP, Simmons EH, Stripius D (1983) Incidence and severity fixation of thoracic spinal deformities using an endoscopic ap- of back pain in adult idiopathic scoliosis. Evans SC, Edgar MA, Hall-Craggs MA, Powell MP, Taylor B, Nor- adaptations in erector spinae muscles in thoracal scoliosis. Fabry G, Van Melkebeek J, Bockx E (1989) Back pain after Har- pathic scoliosis. Gepstein R, Leitner Y, Zohar E, Angel I, Shabat S, Pekarsky I, Friesem pathic scoliosis on the adolescent female.
For many treadmill protocols order super p-force 160 mg line, an increase Energy expenditure for activities such as eating 160 mg super p-force amex, in VO2 <0. BASIC CONCEPTS IN AEROBIC AND VO2peak: When an exercise tests is terminated and the ANAEROBIC EXERCISE criteria described are not met, the higher VO2 achieved is referred to as VO2peak. MAXIMAL AEROBIC POWER Ventilatory threshold: Ventilatory threshold is the point where VE begins to increase disproportionately Maximal aerobic power, or VO , is the greatest to VO2 during incremental exercise testing. It is a 2max measure of “excess” ventilation and has been termed amount of O2 a person can consume during physical exercise. HR at sub- and O2 transport systems, and is considered “power” maximal work rates can be plotted against VO2 and then because it is a rate: L of O2/min. Cycle tests are most appropriate because there are expected VO2 values as a function of Watts (see ing for body weight would yield values of 60 and 45. If person one weighed 70 kg nonexercise data can also be used to estimate VO2max. DETERMINANTS OF AND FACTORS AFFECTING VO2MAX TESTING FOR MAXIMAL AEROBIC POWER Intrinsic and extrinsic factors: Intrinsic factors The best tests for measuring VO2max are incremental affecting VO2max include genetics, gender, body com- tests. A number of issues and concepts are important position/muscle mass, age, and existing pathologies. In addi- Determinants: All systems serving a role in the deliv- tion, the test conditions should be standardized and the ery of O2 can affect VO2max. Central factors include car- diac output, pulmonary ventilation, arterial pressure, test should be tolerated by most people. Motivation hemoglobin (Hb) content, O2 diffusion into and through should not be a major factor, and little to no skill should the lungs, the alveolar ventilation: perfusion ratio, and be required. The primary ways to assess aerobic power are by treadmill walking/running, cycle or arm ergom- etry, and step tests. The test protocol should be incre- mental or progressively increasing work so a true TABLE 8-5 Expected VO2 Values at Designated Power Outputs Between 1 and 3 Min with Cycle Ergometry VO2max is achieved. Different values will be obtained when the mode of exercise changes, and the absolute POWER (W) OXYGEN UPTAKE (L/MIN) value will reflect the muscle mass involved. The leveling off or plateauing effect CHAPTER 8 BASICS IN EXERCISE PHYSIOLOGY 43 Hb-O2 affinity. Peripheral determinants include Onset of blood lactate accumulation: At specific muscle blood flow, capillary density, O2 diffusion to exercise intensity, muscle lactate production exceeds and extraction by muscle cells, Hb-O2 affinity, and utilization and blood lactate begins to accumulate skeletal muscle fiber profiles. Wa, MLSS, and onset of blood lactate accumulation (OBLA) may all demarcate the transition between the heavy and severe exercise domains. AEROBIC AND ANAEROBIC EXERCISE Steady state exercise: When rate of lactate produc- tion is balanced by the rate of oxidative removal and EXERCISE DOMAINS VO2 is stabilized within 3 to 6 min. As such, cardiac Three specific exercise domains were reported by output, HR, and pulmonary gas exchange are in a Gaesser and Poole (1996). Graphical presentations of steady state and exercise can continue for an extended the domains (moderate, heavy, and severe) are pre- period of time. In panel in VO2 beyond the 3rd min is observed when exercise is one, the lactate threshold (TLac) represents the bound- above the lactate threshold. The upper boundary of the heavy gradually increases until it reaches a steady state. TLac represents the lactate threshold and Wa represents critical power or work rate where maximal lactate at steady state occurs. This term, O2 debt, was coined lifted, and is expressed as a percent of the maximum by AV Hill in the early 1900s, but is transitioning to weight (1RM). If the 1RM for a particular exercise excess postexercise oxygen consumption (see is 80 kg, then a weight of 40 kg would be a 50% and below). It is The specificity principle states that physiological, neu- highly correlated with exercise intensity, and the fast rological, and psychological adaptations to training are portion may reflect resynthesis of stored PC and specific to the “imposed demand. The slow develop speed, power, and specific metabolic path- component may reflect elevated body temperature, ways, the imposed demand must target those specific catecholamines, accelerated metabolism (conversion areas. Low numbers of repetitions (6–10 RM) are associated with increases in strength and high num- Resistance exercise is used to improve muscular fit- bers (20–100 RM) are associated with increases in ness, which is a combination of strength, endurance, endurance. Strength is the greatest force a muscle can tion from strength to endurance. The primary components to muscle hypertrophy include a neural response, followed by an upregulation WEIGHT TRAINING PARAMETERS of second messenger systems to activate the family of W hen training with weights, the magnitudes of immediate early genes that dictate the responses of increase in muscle strength and endurance depend on contractile protein genes, and message passing down the specific training parameters: repetitions, sets, to alter protein expression. The Repetition maximum: The amount of force a subject new contractile proteins appear to be incorporated can lift a given number of repetitions defines repeti- into existing myofibrils and there may be a limit to tion maximum (RM). For example, 1RM is the maxi- how large a myofibril can become: they may split at mal force a subject can lift with one repetition and some point. Hypertrophy results primarily from 5RM would be the maximal force someone could lift growth of each muscle cell, rather than an increase in five times. For examples, repetitions could be 5, 10, Physiologic adaptations and performance are linked 12, 25, or 50. For BIOMECHANICAL FACTORS IN MUSCLE STRENGTH example, a training session could consist of three sets Neural control, muscle cross-sectional area, arrange- of 12 repetitions. For example, if the ity, strength-to-mass ratio, body size, joint motion session was three sets of 12 repetitions, the volume (joint mobility, dexterity, flexibility, limberness, and would be 3 × 12 or 36 repetitions. Volume indicates range of motion), point of tendon insertion, and the how much work was done: the greater the volume, the interactions of these factors influence muscle greater the total work. CHAPTER 8 BASICS IN EXERCISE PHYSIOLOGY 45 DELAYED-ONSET MUSCLE SORENESS different VO2max values. Tom would be working at Delayed-onset muscle soreness (DOMS) is a term 2. It is usually noted the day after the exercise and may ADAPTATIONS TO TRAINING last 3 to 4 days. The force generated RESISTANCE TRAINING by a lengthening contraction (eccentric) can be Resistance training induces a variety of adaptations, markedly increased if it is followed by a shortening with clear increases in strength.
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